Published on May 29th, 2011 | by LeW
How to read a CTG
What is Cardiotocography?
Cardiotocography (CTG) is used in pregnancy to monitor both the foetal heart as well as the contractions of the uterus. It is usually only used in the 3rd trimester. It’s purpose is to monitor foetal well-being & allow early detection of foetal distress. An abnormal CTG indicates the need for more invasive investigations & ultimately may lead to emergency caesarian section.
How it works
The device used in cardiotocography is known as a cardiotocograph.
It involves the placement of 2 transducers on the abdomen of a pregnant women.
One transducer records the foetal heart rate using ultrasound.
The other transducer monitors the contractions of the uterus.
It does this by measuring the tension of the maternal abdominal wall.
This provides an indirect indication of intrauterine pressure.
The CTG is then assessed by the midwife & obstetric medical team.
How to read a CTG
To interpret a CTG you need a structured method of assessing it’s various characteristics.
The most popular structure can be remembered using the acronym DR C BRAVADO
DR – Define Risk
C – Contractions
BRa – Baseline Rate
V – Variability
A – Accelerations
D – Decelerations
O - Overall impression
You first need to assess if this pregnancy is high or low risk
This is important as it gives more context to the CTG reading
e.g. If the pregnancy is high risk, your threshold for intervening may be lowered
Reasons a pregnancy may be considered high risk are shown below¹
Maternal medical illness
Previous cesarean section
Intrauterine growth restriction
Premature rupture of the membranes
Oxytocin induction/augmentation of labor
Other risk factors
No prenatal care
Record the number of contractions present in a 10 minute period - e.g. 3 in 10
Each big square is equal to 1 minute, so you look how many contractions occurred in 10 squares
Individual contractions are seen as peaks on the part of the CTG monitoring uterine activity
You should assess contractions for the following:
- Duration – how long do the contractions last?
- Intensity – how strong are the contractions? (assessed using palpation)
In this example there are 2-3 contractions in a 10 minute period - e.g. 3 in 10
Baseline rate of foetal heart
The baseline rate is the average heart rate of the foetus in a 10 minute window
Look at the CTG & assess what the average heart rate has been over the last 10 minutes
Ignore any Accelerations or Decelerations
A normal foetal heart rate is between 110-150 bpm¹
Foetal tachycardia is defined as a baseline heart rate greater than 160 bpm
It can be caused by:¹
- Foetal hypoxia
- Chorioamnionitis – if maternal fever also present
- Foetal or Maternal Anaemia
- Foetal tachyarrhythmia
Foetal bradycardia is defined as a baseline heart rate less than 120 bpm.
Mild bradycardia of between 100-120bpm is common in the following situations:
- Post-date gestation
- Occiput posterior or transverse presentations
Severe prolonged bradycardia (< 80 bpm for > 3 minutes) indicates severe hypoxia
Causes of prolonged severe bradycardia are:¹
- Prolonged cord compression
- Cord prolapse
- Epidural & Spinal Anaesthesia
- Maternal seizures
- Rapid foetal descent
If the cause cannot be identified and corrected, immediate delivery is recommended
Baseline variability refers to the variation of foetal heart rate from one beat to the next
Variability occurs as a result of the interaction between the nervous system, chemoreceptors, barorecptors & cardiac responsiveness.
Therefore it is a good indicator of how healthy the foetus is at that moment in time.
This is because a healthy foetus will constantly be adapting it’s heart rate to respond to changes in it’s environment.
Normal variability is between 10-25 bpm³
To calculate variability you look at how much the peaks & troughs of the heart rate deviate from the baseline rate (in bpm)
Variability can be categorised as: 4
- Reassuring – ≥ 5 bpm
- Non-reassuring – < 5bpm for between 40-90 minutes
- Abnormal – < 5bpm for >90 minutes
Reduced variability can be caused by: ³
- Foetus sleeping - this should last no longer than 40 minutes – most common cause
- Foetal acidosis (due to hypoxia) – more likely if late decelerations also present
- Foetal tachycardia
- Drugs – opiates, benzodiazipine’s, methyldopa, magnesium sulphate
- Prematurity – variability is reduced at earlier gestation (<28 weeks)
- Congenital heart abnormalities
Accelerations are an abrupt increase in baseline heart rate of >15 bpm for >15 seconds
The presence of accelerations is reassuring
Antenatally there should be at least 2 accelerations every 15 minutes¹
Accelerations occurring alongside uterine contractions is a sign of a healthy foetus
However the absence of accelerations with an otherwise normal CTG is of uncertain significance
Decelerations are an abrupt decrease in baseline heart rate of >15 bpm for >15 seconds
There are a number of different types of decelerations, each with varying significance
Early decelerations start when uterine contraction begins & recover when uterine contraction stops
This is due to increased foetal intracranial pressure causing increased vagal tone
It therefore quickly resolves once the uterine contraction ends & intracranial pressure reduces
This type of deceleration is therefore considered to be physiological & not pathological³
Variable decelerations are seen as a rapid fall in baseline rate with a variable recovery phase
They are variable in their duration & may not have any relationship to uterine contractions
They are most often seen during labour & in patients with reduced amniotic fluid volume
Variable decelerations are usually caused by umbilical cord compression¹
- The umbilical vein is often occluded first causing an acceleration in response
- Then the umbilical artery is occluded causing a subsequent rapid deceleration
- When pressure on the cord is reduced another acceleration occurs & then the baseline rate returns
- Accelerations before & after a variable deceleration are known as the “shoulders of deceleration”
- There presence indicates the foetus is not yet hypoxic & is adapting to the reduced blood flow.
Variable decelerations can sometimes resolve if the mother changes position
The presence of persistent variable decelerations indicates the need for close monitoring
Variable decelerations without the shoulders is more worrying as it suggests the foetus is hypoxic
Late decelerations begin at the peak of uterine contraction & recover after the contraction ends.
This type of deceleration indicates there is insufficient blood flow through the uterus & placenta
As a result blood flow to the foetus is significantly reduced causing foetal hypoxia & acidosis
Reduced utero-placental blood flow can be caused by: ¹
- Maternal hypotension
- Uterine hyper-stimulation
The presence of late decelerations is taken seriously & foetal blood sampling for pH is indicated
If foetal blood pH is acidotic it indicates significant foetal hypoxia & the need for emergency C-section
A deceleration that last more than 2 minutes
If it lasts between 2-3 minutes it is classed as Non-Reasurring
If it lasts longer than 3 minutes it is immediately classed as Abnormal
Action must be taken quickly – e.g. Foetal blood sampling / emergency C-section
This type of pattern is rare, however if present it is very serious
It is associated with high rates of foetal morbidity & mortality ¹
It is described as:
- A smooth, regular, wave-like pattern
- Frequency of around 2-5 cycles a minute
- Stable baseline rate around 120-160 bpm
- No beat to beat variability
A sinusoidal pattern indicates:
- Severe foetal hypoxia
- Severe foetal anaemia
- Foetal/Maternal Haemorrhage
Immediate C-section is indicated for this kind of pattern.
Outcome is usually poor
Once you have assessed all aspects of the CTG you need to give your overall impression
The overall impression can be described as either: 4
The overall impression is determined by how many of the CTG features were either reassuring, non-reassuring or abnormal. The NICE guideline below demonstrates how to decide which category a CTG falls into.4
Click to show
3. Clinical obstetrics & gynaecology. 2nd Edition. 2009. B.Magowan, Philip Owen, James Drife
4. Nice guidelines http://www.nice.org.uk/nicemedia/live/11837/36273/36273.pdf